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A human iPSC model of Ligase IV deficiency reveals an important role for NHEJ-mediated-DSB repair in the survival and genomic stability of induced pluripotent stem cells and emerging haematopoietic progenitors.

Tilgner, K.; Neganova, I.; Moreno-Gimeno, I.; Al-Aama, J.Y.; Burks, D.; Yung, S.; Singhapol, C.; Saretzki, G.; Evans, J.; Gorbunova, V.; Gennery, A.; Przyborski, S.; Stojkovic, M.; Armstrong, L.; Jeggo, P.; Lako, M.

Authors

K. Tilgner

I. Neganova

I. Moreno-Gimeno

J.Y. Al-Aama

D. Burks

S. Yung

C. Singhapol

G. Saretzki

J. Evans

V. Gorbunova

A. Gennery

M. Stojkovic

L. Armstrong

P. Jeggo

M. Lako



Citation

Tilgner, K., Neganova, I., Moreno-Gimeno, I., Al-Aama, J., Burks, D., Yung, S., …Lako, M. (2013). A human iPSC model of Ligase IV deficiency reveals an important role for NHEJ-mediated-DSB repair in the survival and genomic stability of induced pluripotent stem cells and emerging haematopoietic progenitors. Cell Death & Differentiation, 20(8), 1089-1100. https://doi.org/10.1038/cdd.2013.44

Journal Article Type Article
Publication Date 2013
Deposit Date Jun 2, 2014
Journal Cell Death and Differentiation
Print ISSN 1350-9047
Electronic ISSN 1476-5403
Publisher Springer Nature
Peer Reviewed Peer Reviewed
Volume 20
Issue 8
Pages 1089-1100
DOI https://doi.org/10.1038/cdd.2013.44
Public URL https://durham-repository.worktribe.com/output/1427837